Fig. 2
From: Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability
LPS induce A549 hyperpermeability and cytoskeleton rearrangement. A549 cells were stimulated by LPS in (a, c) time- or (b, d) dose-dependent manner, and (a, b) TER value and (c, d) permeability coefficient for dextran (Pd) in dextran trans-epithelial flux were evaluated. (e) A549 were treated with or without 100 ng/mL LPS and the image of rhodamine-phalloidin stained F-actin was obtained through a laser confocal microscopy; (f) Fluorescence intensity of F-actin was qualified. n = 3, *P < 0.05 versus control