Combination of clinical symptoms and blood biomarkers can improve discrimination between bacterial or viral community-acquired pneumonia in children

Table 1 Characteristics of children with community-acquired pneumonia (cases) and healthy children (controls), Perth, Western Australia, May 2015 – October 2017

Parameter	Case (%) (N = 230)
Demographic and clinical data
Age
●< 12 months	21 (9.1)
●1–5 years	126 (54.7)
●6–9 years	60 (26.1)
●10+ years	23 (10)
●Male sex	120 (52.1)
●Aboriginal	21 (9.1)
●Premature	32 (13.9)
Smoker present in household	38 (16.5)
Existing health conditions
●Any co-morbidity	34 (14.7)
●Immunodeficiency^a	7 (3.0)
●immunocompromised condition	5 (2.1)
●congenital abnormality^b	17 (7.3)
●chronic respiratory illness	9 (3.9)
●chronic neuromuscular disorder illness	9 (3.9)
●Other^c	1 (0.4)

a: IgG subclass deficiency (n = 1); Low IgA (n = 1); T-cell deficiency (n = 1), Mannose-binding lectin deficiency (n = 2), DiGeorge’s syndrome (n = 2)
b: Capillary malfunction syndrome (n = 1); Beckwith-Wiedemann syndrome and congenital hypothoroidism (n = 2); Atrioventricular septal defect (n = 1); Down syndrome (n = 3); Sotos syndrome (n = 1); Spinal muscular atrophy type 2 (n = 1); Developmental delay (n = 1); Congenital heart disease (n = 2); Prader Willi syndrome (n = 1); Congenital sensorineural deafness (n = 1); Ehlers-Danlos syndrome (n = 1); Gasroschisis (n = 1); Cleft lip (n = 1);
c: Intracranial shunt (n = 1)

ISSN: 1471-2466