Fig. 3From: Baicalin attenuates bleomycin-induced pulmonary fibrosis via adenosine A2a receptor related TGF-β1-induced ERK1/2 signaling pathwayA2aR and baicalin ameliorated pulmonary ultrastructural changes in the BLM mouse model. The ultrathin sections were observed under a Hitachi H-7500 transmission electron microscopy (10,000×). Control groups show little change in ultrastructure; model groups indicate destruction of alveolar epithelial cells and deposition of collagen fibers; baicalin groups exhibit obvious ultrastructural improvement. The ultrastructure of KO mice is more severely damaged. Scale bars represent 1 μmBack to article page